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This meeting took place in 2015
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Granulomas in Infectious and Non-Infectious Diseases (J4)
Organizer(s) Thomas A. Wynn, Paul Kaye and Vishva M. Dixit
January 22—27, 2015
Santa Fe Community Convention Center • Santa Fe, NM USA
Discounted Abstract Deadline: Sep 29, 2014
Abstract Deadline: Oct 22, 2014
Scholarship Deadline: Sep 29, 2014
Discounted Registration Deadline: Nov 20, 2014
Sponsored by Genentech, Inc.
Joint Meeting:
Host Response in Tuberculosis (J3)
Summary of Meeting:
This meeting will address the basic mechanisms of granulomatous inflammation and will focus on several chronic inflammatory diseases in which persistent granuloma formation is the central pathogenic mechanism of disease. The meeting will run in parallel with another on “Host Response in Tuberculosis,” and will include two joint plenary sessions where both audiences will meet together. Granulomas form when the immune system attempts to wall-off substances that it perceives as foreign but is unable to eliminate. These substances include infectious organisms such as bacteria and fungi as well as other known foreign materials. In some cases, however, the offending antigen is unknown as in sarcoidosis. A granuloma is therefore a special type of inflammation, typically an organized collection of macrophages that occurs in a wide variety of infectious and non-infectious diseases including schistosomiasis, histoplasmosis, cryptococcosis, Crohn’s disease, sarcoidosis and rheumatoid arthritis. The goal of this Keystone Symposia meeting is to bring together researchers, clinicians and members of the pharmaceutical industry to discuss the basic mechanics of granuloma formation, elucidate common pathogenic mechanisms and identify novel areas of therapeutic intervention for the large number of chronic granulomatous diseases.
View Scholarships/Awards
This meeting will address the basic mechanisms of granulomatous inflammation and will focus on several chronic inflammatory diseases in which persistent granuloma formation is the central pathogenic mechanism of disease. The meeting will run in parallel with another on “Host Response in Tuberculosis,” and will include two joint plenary sessions where both audiences will meet together. Granulomas form when the immune system attempts to wall-off substances that it perceives as foreign but is unable to eliminate. These substances include infectious organisms such as bacteria and fungi as well as other known foreign materials. In some cases, however, the offending antigen is unknown as in sarcoidosis. A granuloma is therefore a special type of inflammation, typically an organized collection of macrophages that occurs in a wide variety of infectious and non-infectious diseases including schistosomiasis, histoplasmosis, cryptococcosis, Crohn’s disease, sarcoidosis and rheumatoid arthritis. The goal of this Keystone Symposia meeting is to bring together researchers, clinicians and members of the pharmaceutical industry to discuss the basic mechanics of granuloma formation, elucidate common pathogenic mechanisms and identify novel areas of therapeutic intervention for the large number of chronic granulomatous diseases.
View Scholarships/Awards
No registration fees are used to fund entertainment or alcohol at this conference
THURSDAY, JANUARY 22
FRIDAY, JANUARY 23
SATURDAY, JANUARY 24
SUNDAY, JANUARY 25
MONDAY, JANUARY 26
TUESDAY, JANUARY 27
Conference Program Print | View meeting in 12 hr (am/pm) time
THURSDAY, JANUARY 22
18:00—20:00
Welcome Mixer
No registration fees are used to fund alcohol served at this function.
08:00—09:00
Keynote Address
*
Thomas A. Wynn,
Pfizer, USA
Alberto Mantovani,
Humanitas University, Italy
Macrophage Plasticity and Polarization in Granulomatous Inflammation
Macrophage Plasticity and Polarization in Granulomatous Inflammation
08:00—09:00
Keynote Address
Setting the stage for addressing tuberculosis from a human perspective--from basic to clinical research.
*
JoAnne L. Flynn,
University of Pittsburgh School of Medicine, USA
09:00—11:15
Granulomas Associated with Type-1 Immunity
Paul Kaye,
University of York, UK
Understanding Granuloma Heterogeneity in Visceral Leishmaniasis
Understanding Granuloma Heterogeneity in Visceral Leishmaniasis
Martin Rottenberg,
Karolinska Institutet, Sweden
SOCS3 in either Myeloid or T Cells Conveys Resistance to Infection with Mycobacterium tuberculosis
SOCS3 in either Myeloid or T Cells Conveys Resistance to Infection with Mycobacterium tuberculosis
Jean-Laurent Casanova,
Rockefeller University, USA
Toward a Genetic Theory of Childhood Infectious Diseases
Toward a Genetic Theory of Childhood Infectious Diseases
Evelina Guirado,
Ohio State University, USA
Short Talk: Characterization of Host and Microbial Determinants in Individuals with Latent Tuberculosis Infection Using a Human Granuloma Model
Short Talk: Characterization of Host and Microbial Determinants in Individuals with Latent Tuberculosis Infection Using a Human Granuloma Model
09:00—11:15
Immunity in the Lung
Events in the airway and interactions with early cells influence outcome.
*
Alan Sher,
NIAID, National Institutes of Health, USA
Henry Charles Mwandumba,
Liverpool School of Tropical Medicine, UK
TB/HIV Interactions in the Airways
TB/HIV Interactions in the Airways
Gerhard Walzl,
Stellenbosch University, South Africa
Human TB Treatment Response Studies Using PET/CT Imaging: Inconvenient Observations
Human TB Treatment Response Studies Using PET/CT Imaging: Inconvenient Observations
Wendy A. Burgers,
University of Cape Town, South Africa
Short Talk: Defects in Multiple Mycobacterial T Helper Subsets in Blood and Lungs in Early HIV Infection
Short Talk: Defects in Multiple Mycobacterial T Helper Subsets in Blood and Lungs in Early HIV Infection
14:30—16:30
Workshop 1: Basic Biology
*
Matyas Sandor,
University of Wisconsin, USA
Matthew McPeek,
East Carolina University, USA
MicroRNAs Targeting PPAR? Pathways Are Elevated in Bronchoalveolar Lavage (BAL) Cells from Sarcoidosis Patients and from Mice Bearing Carbon Nanotube Induced Granulomas
MicroRNAs Targeting PPAR? Pathways Are Elevated in Bronchoalveolar Lavage (BAL) Cells from Sarcoidosis Patients and from Mice Bearing Carbon Nanotube Induced Granulomas
Marie Lipoldova,
Institute of Molecular Genetics, Czech Republic
Analysis of Granuloma Formation and Characteristics Using Defined Genomic Constructs
Analysis of Granuloma Formation and Characteristics Using Defined Genomic Constructs
Deepak Kaushal,
Texas Biomedical Research Institute, USA
Role of Sensor Kinase DosS in Virulence of Mycobacterium tuberculosis in C3HeB/FeJ Mice with Classical Granulomatous Lesions
Role of Sensor Kinase DosS in Virulence of Mycobacterium tuberculosis in C3HeB/FeJ Mice with Classical Granulomatous Lesions
Teresa A. Hudock,
Tulane National Primate Research Center, USA
Transcriptome Analysis of Mycobacterium tuberculosis in Primate Lung Granulomas
Transcriptome Analysis of Mycobacterium tuberculosis in Primate Lung Granulomas
Albert Byungyun Jeon,
Colorado State University, USA
Reversal of Phenotypic and Genotypic Antimicrobial Drug Resistance in Mycobacterium tuberculosis with 2 Aminoimadazole-Based Small Molecule Adjuvants
Reversal of Phenotypic and Genotypic Antimicrobial Drug Resistance in Mycobacterium tuberculosis with 2 Aminoimadazole-Based Small Molecule Adjuvants
14:30—16:30
Workshop: Novel Approaches to Treatment of Tuberculosis
*
Christopher M. Sassetti,
University of Massachusetts Medical School, USA
*
Brian C. VanderVen,
Cornell University, USA
Chemical Screening against Mycobacterium tuberculosis in Macrophages Identifies Inhibitors of Cholesterol Utilization
Chemical Screening against Mycobacterium tuberculosis in Macrophages Identifies Inhibitors of Cholesterol Utilization
Frederick K. Balagaddé,
K-RITH KwaZulu-Natal Research Institute for TB & HIV, South Africa
Confinement-Induced Drug-Tolerance in Microfluidic Bioreactors
Confinement-Induced Drug-Tolerance in Microfluidic Bioreactors
Robert Blomgran,
Linköping University, Sweden
Pharmacological Inhibition of mTORC1 Is Not the Treatment for HIV Mycobacterium tuberculosis Co-Infection
Pharmacological Inhibition of mTORC1 Is Not the Treatment for HIV Mycobacterium tuberculosis Co-Infection
Meenal Datta,
Massachusetts General Hospital/Harvard Medical School, USA
Anti-VEGF Treatment Normalizes Tuberculosis Granuloma Vasculature and Improves Small Molecule Delivery
Anti-VEGF Treatment Normalizes Tuberculosis Granuloma Vasculature and Improves Small Molecule Delivery
Scott M. Irwin,
Colorado State University, USA
Bedaquiline and its Metabolite Display Reduced Penetration into Caseous Necrotic Pulmonary Lesions in C3HeB/FeJ Mice
Bedaquiline and its Metabolite Display Reduced Penetration into Caseous Necrotic Pulmonary Lesions in C3HeB/FeJ Mice
Ekta Lachmandas,
Radboud University Medical Center, Netherlands
Rewiring of Cellular Metabolism via the AKT/mTOR Pathway Forms the Basis of Host Defense against Mycobacterium tuberculosis
Rewiring of Cellular Metabolism via the AKT/mTOR Pathway Forms the Basis of Host Defense against Mycobacterium tuberculosis
Tige R. Rustad,
Seattle BioMed, USA
The Wiring Diagram of Mycobacterium tuberculosis: Generating and Using an Experimentally-Derived Transcriptional Regulatory Map
The Wiring Diagram of Mycobacterium tuberculosis: Generating and Using an Experimentally-Derived Transcriptional Regulatory Map
Suraj P. Parihar,
University of Cape Town, South Africa
Role of Statins against Mycobacterium tuberculosis Infection
Role of Statins against Mycobacterium tuberculosis Infection
17:00—19:00
Infectious and Non-Infectious Granulomatous Disease
*
Paul Kaye,
University of York, UK
Margherita T. Cantorna,
Pennsylvania State University, USA
The Implications of Vitamin D Status on T Cells, the Microbiome and Crohn’s Disease
The Implications of Vitamin D Status on T Cells, the Microbiome and Crohn’s Disease
R Balfour Balfour Sartor,
University of North Carolina - Chapel Hill, USA
Granulomatous Enterocolitis and Inflammation in Crohn's Disease: Intersection of Functionally Abnormal Bacteria and Defective Innate and Adaptive Immune Responses
Granulomatous Enterocolitis and Inflammation in Crohn's Disease: Intersection of Functionally Abnormal Bacteria and Defective Innate and Adaptive Immune Responses
Andreea Geamanu,
Wayne State University, USA
Short Talk: Metabolomics Links Alterations in Fatty Acid Metabolism, Inflammation and Gut Microbiota in Sarcoidosis
Short Talk: Metabolomics Links Alterations in Fatty Acid Metabolism, Inflammation and Gut Microbiota in Sarcoidosis
Damien J.C. Montamat-Sicotte,
McGill Research Institute RI-MUHC, Canada
Short Talk: NOD2: Innate Immunity, Bacterial Infection and Chronic Inflammation
Short Talk: NOD2: Innate Immunity, Bacterial Infection and Chronic Inflammation
17:00—19:00
Early Events after TB Infection
Early events in the infectious process.
*
Henry Charles Mwandumba,
Liverpool School of Tropical Medicine, UK
Thomas R. Hawn,
University of Washington, USA
Innate Immunogenetics of TB in Humans
Innate Immunogenetics of TB in Humans
Ludovic P. Desvignes,
New York University School of Medicine, USA
Short Talk: Dynamics of Early Growth and Spread of Mycobacterium tuberculosis in vivo Reveal Sequential Infection of Myeloid Cell Populations
Short Talk: Dynamics of Early Growth and Spread of Mycobacterium tuberculosis in vivo Reveal Sequential Infection of Myeloid Cell Populations
08:00—11:30
Pathogenic Mechanisms in Chronic Granulomatous Disease (Joint)
*
Vishva M. Dixit,
Genentech, Inc., USA
Gilla Kaplan,
Bill & Melinda Gates Foundation, USA
Lesion-Specific Immune Activation in Granulomas of Patients with Pulmonary Tuberculosis
Lesion-Specific Immune Activation in Granulomas of Patients with Pulmonary Tuberculosis
Thomas A. Wynn,
Pfizer, USA
Mechanisms of Fibrosis
Mechanisms of Fibrosis
Lalita Ramakrishnan,
University of Cambridge, UK
Mechanisms and Consequences of Tuberculous Granuloma Necrosis
Mechanisms and Consequences of Tuberculous Granuloma Necrosis
Shahin Shafiani,
Center for Infectious Disease Research, USA
Short Talk: Foxp3+ Regulatory T Cells Are Host-Protective during Chronic Tuberculosis and Promote a Pathogen-Specific Immune Response
Short Talk: Foxp3+ Regulatory T Cells Are Host-Protective during Chronic Tuberculosis and Promote a Pathogen-Specific Immune Response
Philana Ling Lin,
University of Pittsburgh, USA
Short Talk: Spatial Patterns of Granuloma Development Differ between Infection and Reactivation
Short Talk: Spatial Patterns of Granuloma Development Differ between Infection and Reactivation
Igor B. Kramnik,
Boston University, USA
Short Talk: Necrosis in Granulomas: Mechanism and Therapeutic Approaches
Short Talk: Necrosis in Granulomas: Mechanism and Therapeutic Approaches
17:00—19:00
Role of Myeloid Cells in Granuloma Formation
*
P'ng Loke,
New York University School of Medicine, USA
Peter J. Murray,
Max-Planck-Institut für Biochemie, Germany
Amino Acid Metabolism by Innate Immune Cells in Granulomatous Inflammation
Amino Acid Metabolism by Innate Immune Cells in Granulomatous Inflammation
Frederic Geissmann,
Memorial Sloan Kettering Cancer Center, USA
Langerhans Cell Histiocytosis, a Pediatric Granulomatous Disease
Langerhans Cell Histiocytosis, a Pediatric Granulomatous Disease
Keke C. Fairfax,
University of Utah, USA
Short Talk: Schistosoma mansoni Infection Induces Anti-Atherogenic Transcriptional Changes in Hepatic Macrophages
Short Talk: Schistosoma mansoni Infection Induces Anti-Atherogenic Transcriptional Changes in Hepatic Macrophages
17:00—19:00
Bacterial Interactions with Host Cells
Mycobacterium tuberculosis interactions with host
*
Jennifer Philips,
Washington University School of Medicine, USA
Sarah M. Fortune,
Harvard TH Chan School of Public Health, USA
Dissecting Bacterial Survival, Replication and Mutation in the Host Environment
Dissecting Bacterial Survival, Replication and Mutation in the Host Environment
W. Henry Boom,
Case Western Reserve University, USA
Evasion of T Cell Immunity by Mycobacterium tuberculosis
Evasion of T Cell Immunity by Mycobacterium tuberculosis
Christopher M. Sassetti,
University of Massachusetts Medical School, USA
Systematic Genetic Approaches to Understand TB Pathogenesis
Systematic Genetic Approaches to Understand TB Pathogenesis
Jordi B. Torrelles,
Ohio State University, USA
Short Talk: Lung Mucosa Lining Fluid Modifies Mycobacterium tuberculosis to Reprogram Neutrophil Killing Mechanisms to Preserve the Anti-Inflammatory Integrity of the Lung
Short Talk: Lung Mucosa Lining Fluid Modifies Mycobacterium tuberculosis to Reprogram Neutrophil Killing Mechanisms to Preserve the Anti-Inflammatory Integrity of the Lung
19:00—20:00
Social Hour with Lite Bites
No registration fees are used to fund alcohol served at this function.
08:00—11:00
Granulomas Associated with Type-2 Immunity
*
Padmini Salgame,
Rutgers University, USA
Edward J. Pearce,
Max Planck Institute of Immunobiology and Epigenetics, Germany
Metabolic Reprogramming of Myeloid Cells during Granuloma Formation
Metabolic Reprogramming of Myeloid Cells during Granuloma Formation
William C. Gause,
Rutgers New Jersey Medical School, USA
Regulation of Type-2 Inflammation and Pathology in the Lung and Gut during Nematode Infection
Regulation of Type-2 Inflammation and Pathology in the Lung and Gut during Nematode Infection
P'ng Loke,
New York University School of Medicine, USA
Alternatively Activated Macrophages during Granuloma Formation in Schistosomiasis
Alternatively Activated Macrophages during Granuloma Formation in Schistosomiasis
Jessica C. Jang,
University of California, Riverside, USA
Short Talk: Human Resistin Is Induced in Multiple Helminth Infections and Promotes Proinflammatory Cytokines and Monocyte-Rich Lung Granulomas
Short Talk: Human Resistin Is Induced in Multiple Helminth Infections and Promotes Proinflammatory Cytokines and Monocyte-Rich Lung Granulomas
08:00—11:15
Risk of TB Disease
Factors that influence risk of disease
*
Tom H. M. Ottenhoff,
Leiden University Medical Center, Netherlands
Thomas J. Scriba,
University of Cape Town, South Africa
Prospective Correlates of Risk of TB Disease
Prospective Correlates of Risk of TB Disease
Katrin D. Mayer-Barber,
NIAID, National Institutes of Health, USA
Inflammatory Cytokine Networks during TB
Inflammatory Cytokine Networks during TB
January Weiner,
Max Planck Institute of Infection Biology, Germany
TB Biomarkers Across Cohorts and Sample Types
TB Biomarkers Across Cohorts and Sample Types
Joel D. Ernst,
University of California, San Francisco, USA
Antigen Conservation and Diversity in Human Tuberculosis
Antigen Conservation and Diversity in Human Tuberculosis
Rustin Lovewell,
University of Massachusetts Medical School, USA
Short Talk: The Role of Neutrophils in Progressive Tuberculosis
Short Talk: The Role of Neutrophils in Progressive Tuberculosis
Diane Joyce Ordway,
Colorado State University, USA
Short Talk: BCG Efficacy in Guinea Pigs Naturally Exposed to Patients with Tuberculosis
Short Talk: BCG Efficacy in Guinea Pigs Naturally Exposed to Patients with Tuberculosis
17:00—19:00
Granulomatous Diseases of the Lung
*
Daniel L. Barber,
NIAID, National Institutes of Health, USA
Randall J. Basaraba,
Colorado State University, USA
Granuloma-Targeted Therapy in the Treatment of Tuberculosis
Granuloma-Targeted Therapy in the Treatment of Tuberculosis
Andrew P. Fontenot,
University of Colorado Denver, USA
Gene-Environment Interactions and the Development of Chronic Beryllium Disease
Gene-Environment Interactions and the Development of Chronic Beryllium Disease
Luigina Romani,
University di Perugia, Italy
Chronic Inflammation in Fungal Diseases: From Basic Science to Therapeutic Intervention
Chronic Inflammation in Fungal Diseases: From Basic Science to Therapeutic Intervention
Bryce C. Asay,
Colorado State University, USA
Short Talk: Heterogeneity in Lesion Types in C3HeB/FeJ Mice Is Modulated by Specific Characteristics of the Mycobacterium tuberculosis Strain Used
Short Talk: Heterogeneity in Lesion Types in C3HeB/FeJ Mice Is Modulated by Specific Characteristics of the Mycobacterium tuberculosis Strain Used
17:00—19:00
B Cells Responses to TB
The neglected lymphocyte in TB
*
Shabaana Khader,
Washington University School of Medicine, USA
John R. Chan,
Albert Einstein College of Medicine, USA
B Cells Promote Granulomatous Inflammation during Chronic Mycobacterium tuberculosis Infection
B Cells Promote Granulomatous Inflammation during Chronic Mycobacterium tuberculosis Infection
Galit Alter,
MIT and Harvard University, USA
A Case for Antibody Fc-Effector Function in Tb Containment
A Case for Antibody Fc-Effector Function in Tb Containment
Alan Bénard,
Institut de Pharmacologie et de Biologie Structurale, France
Short Talk: B Cells Modulate Inflammation during Mycobacterium tuberculosis Infection in a MyD88- and Type I Interferon-Dependent Manner
Short Talk: B Cells Modulate Inflammation during Mycobacterium tuberculosis Infection in a MyD88- and Type I Interferon-Dependent Manner
19:00—20:00
Social Hour with Lite Bites
No registration fees are used to fund alcohol served at this function.
08:00—11:15
Computational Modeling, Imaging and Treatment
*
Andrew P. Fontenot,
University of Colorado Denver, USA
Annemarie H. Meijer,
Leiden University, Netherlands
In vivo Study of Anti-Mycobacterial Autophagy and Granuloma Formation in the Zebrafish Model
In vivo Study of Anti-Mycobacterial Autophagy and Granuloma Formation in the Zebrafish Model
Ronald N. Germain,
NIAID, National Institutes of Health, USA
Live Imaging Anti-Mycobacterial Immunity
Live Imaging Anti-Mycobacterial Immunity
Matyas Sandor,
University of Wisconsin, USA
Mycobacterial Granuloma Dynamics: Repopulation, Reformation and Cellular Traffic
Mycobacterial Granuloma Dynamics: Repopulation, Reformation and Cellular Traffic
Steven M. Holland,
NIAID, National Institutes of Health, USA
Genetics and Treatment of Chronic Granulomatous Disease
Genetics and Treatment of Chronic Granulomatous Disease
Elsje Pienaar,
University of Michigan, USA
Short Talk: Interplay between Mycobacterial Metabolism and Granuloma Dynamics in Tuberculosis
Short Talk: Interplay between Mycobacterial Metabolism and Granuloma Dynamics in Tuberculosis
David M. Tobin,
Duke University School of Medicine, USA
Short Talk: Interception of Host Angiogenic Signaling Limits Mycobacterial Growth
Short Talk: Interception of Host Angiogenic Signaling Limits Mycobacterial Growth
08:00—11:15
Protective Immunity and Vaccines
Protection against TB
*
Thomas Evans,
TomegaVax, USA
Denise Kirschner,
University of Michigan, USA
A Novel Two-Pronged Approach to Biomarker Discovery in Tuberculosis
A Novel Two-Pronged Approach to Biomarker Discovery in Tuberculosis
Willem A. Hanekom,
Bill & Melinda Gates Foundation, USA
Vaccines against TB: Where Are We Going?
Vaccines against TB: Where Are We Going?
D. Branch Moody,
Brigham and Women's Hospital, USA
Building Technology-Based T Cell Response to Non-Polymorphic CD1 Proteins
Building Technology-Based T Cell Response to Non-Polymorphic CD1 Proteins
Robert A. Seder,
NIAID, National Institutes of Health, USA
What Is Needed to Protect Against TB?
What Is Needed to Protect Against TB?
Stephen M. Carpenter,
Regeneron Pharmaceuticals, USA
Short Talk: Heavily-Armed but Outnumbered: A Selective Loss of Memory CD8+ T Cells during Tuberculosis May Eclipse the Benefit of Vaccination
Short Talk: Heavily-Armed but Outnumbered: A Selective Loss of Memory CD8+ T Cells during Tuberculosis May Eclipse the Benefit of Vaccination
Guangwu Xu,
Vaccine & Gene Therapy Inst, and the Oregon National Primate Research Ctr, OHSU, USA
Short Talk: Cytomegalovirus Vector-Based Tuberculosis Vaccines Provide Superior Protection to BCG after Intra-Bronchial mTb Challenge of Indian-Origin Rhesus macaques
Short Talk: Cytomegalovirus Vector-Based Tuberculosis Vaccines Provide Superior Protection to BCG after Intra-Bronchial mTb Challenge of Indian-Origin Rhesus macaques
14:30—16:30
Workshop 2: Clinical and Translational
*
David M. Tobin,
Duke University School of Medicine, USA
Delia Goletti,
National Institute for Infectious Diseases, Italy
Can Blood or Urine IP-10 Discriminate between Active and Non Active Tuberculosis in Children from High Endemic Areas?
Can Blood or Urine IP-10 Discriminate between Active and Non Active Tuberculosis in Children from High Endemic Areas?
Lobelia Samavati,
Wayne State University School of Medicine, USA
Development of a Sarcoidosis Library to Detect Sarcoidosis and Tuberculosis by a Panel of Novel Biomarkers
Development of a Sarcoidosis Library to Detect Sarcoidosis and Tuberculosis by a Panel of Novel Biomarkers
Daniel Torocsik,
University of Debrecen, Hungary
Immunohistochemical Characterization of FXIII-A+ Cells in Non-Infectious Granulomatous Skin Lesions
Immunohistochemical Characterization of FXIII-A+ Cells in Non-Infectious Granulomatous Skin Lesions
Javeed Ali Shah,
University of Washington, USA
TOLLIP Variants Are Associated with Susceptibility to Leprosy and Dermal Expression of TOLLIP and IL-1Ra in Nepal
TOLLIP Variants Are Associated with Susceptibility to Leprosy and Dermal Expression of TOLLIP and IL-1Ra in Nepal
Ho Namkoong,
Keio University School of Medicine, Japan
Clarithromycin Expands CD11b+Gr-1+Cells to Ameliorate Post-Influenza Pneumococcal Pneumonia
Clarithromycin Expands CD11b+Gr-1+Cells to Ameliorate Post-Influenza Pneumococcal Pneumonia
17:00—19:00
TB and Co-Morbidities (Joint)
Interactions of TB with other diseases
*
Willem A. Hanekom,
Bill & Melinda Gates Foundation, USA
Padmini Salgame,
Rutgers University, USA
TB and Worms
TB and Worms
Jovvian George Parakkal,
New York Blood Center-LFKRI, USA
Short Talk: Influence of Helminth Infections on the Innate and Adaptive Immune Responses to Pulmonary Tuberculosis
Short Talk: Influence of Helminth Infections on the Innate and Adaptive Immune Responses to Pulmonary Tuberculosis
19:30—20:30
Social Hour with Lite Bites
No registration fees are used to fund alcohol served at this function.
20:00—23:00
Entertainment
Entertainment is not subsidized by conference registration fees nor any U.S. federal government grants. Funding for this expense is provided by other revenue sources.
*Session Chair †Invited, not yet responded.
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