Santa Fe Community Convention Center Floorplan
Registered Attendees
Registered attendees (and speakers, organizers, etc.) will have access to the following items from their Account page:
- Abstracts from speakers and poster sessions, including the joint meeting abstracts, available 30 days prior to the meeting
(You can edit your own abstract from My Account page as well)
NOTE: Abstract authors/submitters may choose to not have their abstract available online and in the secure mobile app until a week before the meeting.
- Full participant list, including joint meeting participants
- Printable Invoices and Invitation Letters
- Scholarship Information
- Lodging Information
Login to My Account page
This meeting took place in 2014
Here are the related meetings in 2020:
Why So Many Ways to Die? Apoptosis, Necroptosis, Pyroptosis and Beyond (T3)
For a complete list of the meetings for the upcoming/current season, see our meeting list, or search for a meeting.
The Chemistry and Biology of Cell Death (Q6)
Organizer(s) Guy S. Salvesen, Matthew S. Bogyo and Jennie R. Lill
February 18—23, 2014
Santa Fe Community Convention Center • Santa Fe, NM USA
Discounted Abstract Deadline: Oct 17, 2013
Abstract Deadline: Nov 21, 2013
Scholarship Deadline: Oct 17, 2013
Discounted Registration Deadline: Dec 17, 2013
Sponsored by Genentech, Inc. and Infinity Pharmaceuticals, Inc.
Joint Meeting:
Mitochondrial Dynamics and Physiology (Q5)
Summary of Meeting:
Cell death can occur through distinct mechanisms, and the signaling pathways leading to these lethal outcomes are becoming established. Although therapeutic strategies targeting specific cell death mediators were introduced over 10 years ago, the breadth of cell death mechanisms should present numerous additional possibilities for drug development. To better understand the Achilles’ heels for targeted therapy, we need to comprehend the chemistry behind cell death paradigms. Coupling the breadth of chemical biology with genetic analysis of cell death pathways in model organisms should provide not only a comprehensive understanding, but reveal tractable targets. This meeting will address the normal regulation and pathogenic dysfunction of distinct cell death modalities, discuss new modalities for therapeutic intervention and highlight chemical biology efforts that are leading to a better understanding of the role that cell death plays in health and disease. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on “Mitochondrial Dynamics and Physiology,” which will share keynote addresses and a plenary session with this meeting.
View Scholarships/Awards
Cell death can occur through distinct mechanisms, and the signaling pathways leading to these lethal outcomes are becoming established. Although therapeutic strategies targeting specific cell death mediators were introduced over 10 years ago, the breadth of cell death mechanisms should present numerous additional possibilities for drug development. To better understand the Achilles’ heels for targeted therapy, we need to comprehend the chemistry behind cell death paradigms. Coupling the breadth of chemical biology with genetic analysis of cell death pathways in model organisms should provide not only a comprehensive understanding, but reveal tractable targets. This meeting will address the normal regulation and pathogenic dysfunction of distinct cell death modalities, discuss new modalities for therapeutic intervention and highlight chemical biology efforts that are leading to a better understanding of the role that cell death plays in health and disease. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on “Mitochondrial Dynamics and Physiology,” which will share keynote addresses and a plenary session with this meeting.
View Scholarships/Awards
No registration fees are used to fund entertainment or alcohol at this conference
TUESDAY, FEBRUARY 18
WEDNESDAY, FEBRUARY 19
THURSDAY, FEBRUARY 20
FRIDAY, FEBRUARY 21
SATURDAY, FEBRUARY 22
SUNDAY, FEBRUARY 23
Conference Program Print | View meeting in 12 hr (am/pm) time
TUESDAY, FEBRUARY 18
08:00—10:00
Keynote Session (Joint)
*
Guy S. Salvesen,
Sanford-Burnham Medical Research Institute, USA
*
Rodrigue Rossignol,
University of Bordeaux, France
Vishva M. Dixit,
Genentech, Inc., USA
Signaling Lessons from Death Receptors: The Inflammasome and Beyond
Signaling Lessons from Death Receptors: The Inflammasome and Beyond
10:20—12:00
Mitochondria and Death (Joint)
*
Sally A. Kornbluth,
Duke University Medical Center, USA
*
Heidi M. McBride,
McGill University, Canada
David W. Andrews,
Sunnybrook Research Institute, Canada
Shedding Light on the Mechanisms of Action of Bcl-2 Family Proteins
Shedding Light on the Mechanisms of Action of Bcl-2 Family Proteins
Richard J. Youle,
NINDS, National Institutes of Health, USA
Damage Control - How the Pink1/Parkin Pathway Can Regulate Removal of Impaired Mitochondria by Autophagy
Damage Control - How the Pink1/Parkin Pathway Can Regulate Removal of Impaired Mitochondria by Autophagy
Dhyan Chandra,
Roswell Park Comprehensive Cancer Center, USA
Short Talk: Macromolecular Changes on Mitochondria and their Impact on DNA Damage-Induced Apoptotic Cell Death
Short Talk: Macromolecular Changes on Mitochondria and their Impact on DNA Damage-Induced Apoptotic Cell Death
Luca Scorrano,
University of Padova, Italy
Role of Mitochondrial Dynamics in Embryogenesis
Role of Mitochondrial Dynamics in Embryogenesis
17:00—19:00
Fundamental Death Mechanisms
*
John Silke,
Walter and Eliza Hall Institute of Medical Research, Australia
Douglas R. Green,
St. Jude Children's Research Hospital, USA
Apoptotic and Non-Apoptotic Developmental Cell Death in Mice
Apoptotic and Non-Apoptotic Developmental Cell Death in Mice
Andreas E. Strasser,
Walter and Eliza Hall Institute of Medical Research, Australia
The Role of the BCL-2 Regulated (Mitochondrial) Apoptotic Pathway in Morphogenesis during Mouse Development
The Role of the BCL-2 Regulated (Mitochondrial) Apoptotic Pathway in Morphogenesis during Mouse Development
Peter E. Czabotar,
Walter and Eliza Hall Institute of Medical Research, Australia
Crystal Structures of Bax and Bak Reveal Molecular Events Initiating Apoptosis
Crystal Structures of Bax and Bak Reveal Molecular Events Initiating Apoptosis
17:00—19:00
Mitochondrial Architecture
*
David C. Chan,
California Institute of Technology, USA
Thomas Langer,
CECAD Research Center, Germany
Proteolytic Control of Mitochondrial Membrane Dynamics
Proteolytic Control of Mitochondrial Membrane Dynamics
Peter Rehling,
University Medical Centre Göttingen, Germany
Biogenesis of Mitochondrial Membrane Protein Complexes
Biogenesis of Mitochondrial Membrane Protein Complexes
Victoria L. Hewitt,
Medical Research Council, UK
Short Talk: The Role of SAM and ERMES Complexes in Candida albicans Mitochondria
Short Talk: The Role of SAM and ERMES Complexes in Candida albicans Mitochondria
08:00—11:00
Chemical Biology
*
Jennie R. Lill,
Genentech, Inc., USA
Mark B. Hampton,
University of Otago, New Zealand
Reactive Oxygen Species and Cell Death
Reactive Oxygen Species and Cell Death
Matthew S. Bogyo,
Stanford University School of Medicine, USA
A Chemical Biology Approach for the Selective Imaging and Inhibition of Caspases
A Chemical Biology Approach for the Selective Imaging and Inhibition of Caspases
Sarah H. MacKenzie,
North Carolina State University, USA
Short Talk: A Natural Peptide Binds to an Allosteric Site in Caspase-3
Short Talk: A Natural Peptide Binds to an Allosteric Site in Caspase-3
Sharan R. Srinivasan,
University of Michigan, USA
Short Talk: Allosteric Inhibitor of Hsp70 Reveals its Role at the Intersection of Multiple Cell Death Pathways
Short Talk: Allosteric Inhibitor of Hsp70 Reveals its Role at the Intersection of Multiple Cell Death Pathways
Guillaume Lessene,
Walter and Eliza Hall Institute of Medical Research, Australia
Short Talk: Novel, Potent and Selective Inhibitors of the Pro-Survival BCL-2 Family Member BCL-XL
Short Talk: Novel, Potent and Selective Inhibitors of the Pro-Survival BCL-2 Family Member BCL-XL
08:00—11:00
Mitochondrial Dynamics
*
Jodi Nunnari,
University of California, Davis, USA
Heidi M. McBride,
McGill University, Canada
The Physiological Implications of Mitochondrial SUMOylation
The Physiological Implications of Mitochondrial SUMOylation
Gia K. Voeltz,
University of Colorado Boulder, USA
Snapshots of ER-Mediated Mitochondrial Constriction Sites
Snapshots of ER-Mediated Mitochondrial Constriction Sites
Henry N. Higgs,
Geisel School of Medicine at Dartmouth, USA
A Role for Actin, Formins and Myosin II in Mammalian Mitochondrial Fission
A Role for Actin, Formins and Myosin II in Mammalian Mitochondrial Fission
Stefan Strack,
University of Iowa, USA
Regulation of Mitochondrial Fission in Neuronal Development and Synaptic Plasticity
Regulation of Mitochondrial Fission in Neuronal Development and Synaptic Plasticity
Robert A. Screaton,
Sunnybrook Research Institute, Canada
Short Talk: Genome-Wide RNAi Screen Identifies ROMO1 as an Essential Redox-Dependent Regulator of Mitochondrial Dynamics
Short Talk: Genome-Wide RNAi Screen Identifies ROMO1 as an Essential Redox-Dependent Regulator of Mitochondrial Dynamics
14:30—16:30
Workshop 1: Autophagy and Mitophagy
*
Lisa M. Lindqvist,
Walter and Eliza Hall Institute of Medical Research, Australia
Bcl-2, Bcl-xL and Mcl-1 Are Not Major Regulators of Autophagy
Bcl-2, Bcl-xL and Mcl-1 Are Not Major Regulators of Autophagy
Juliane C. Campos,
University of Sao Paulo, Brazil
Disrupted Mitochondrial Dynamics and Impaired Autophagy in Heart Failure: Impact of Exercise Training
Disrupted Mitochondrial Dynamics and Impaired Autophagy in Heart Failure: Impact of Exercise Training
Isabella Caniggia,
Lunenfeld-Tanenbaum Research Institute, Canada
Disruption of Sphingolipid Metabolism Augments Placental Autophagy
Disruption of Sphingolipid Metabolism Augments Placental Autophagy
Aditya Murthy,
Genentech, Inc., USA
A Crohn’s Disease Mutation in the Autophagy Gene Atg16L1 Facilitates its Caspase-Mediated Degradation
A Crohn’s Disease Mutation in the Autophagy Gene Atg16L1 Facilitates its Caspase-Mediated Degradation
Gavin Clive Higgins,
Baker IDI Heart and Diabetes Institute, Australia
Impaired Mitophagy Activity in Experimental Diabetic Nephropathy
Impaired Mitophagy Activity in Experimental Diabetic Nephropathy
Malle Kuum,
University of Tartu, Estonia
Directed Laser Irradiation-Based Method to Study Selective Mitophagy in Neurons
Directed Laser Irradiation-Based Method to Study Selective Mitophagy in Neurons
Baris Bingol,
Genentech, Inc., USA
DUBs Regulate the Parkin/PINK1 Mitophagy Pathway
DUBs Regulate the Parkin/PINK1 Mitophagy Pathway
14:30—16:30
Workshop 1: Mitochondrial Research and Drug Discovery
*
Thomas Langer,
CECAD Research Center, Germany
Ying Liu,
Peking University, China
Endogenous Small Molecule Signals of C. elegans Mitochondrial Dysfunction Couple to the Induction of Detoxification and Pathogen Response Pathways
Endogenous Small Molecule Signals of C. elegans Mitochondrial Dysfunction Couple to the Induction of Detoxification and Pathogen Response Pathways
Bjoern Oettinghaus,
University Hospital Basel, Switzerland
Induced Drp1 Ablation in the Adult Mouse Forebrain
Induced Drp1 Ablation in the Adult Mouse Forebrain
Melissa Vos,
University of Lübeck, Germany
Stimulation of the Electron Transport Chain as a Possible Therapeutic Strategy for Parkinson’s Disease
Stimulation of the Electron Transport Chain as a Possible Therapeutic Strategy for Parkinson’s Disease
Daniel J. Gonzalez-Dunia,
Inserm UMR1043, France
Bornavirus X Protein: A New Tool Against Neurodegenerative Disorders?
Bornavirus X Protein: A New Tool Against Neurodegenerative Disorders?
Simone Caielli,
Baylor Institute for Immunology Research, USA
Incomplete Mitophagy in Human Neutrophils Leads to Extrusion of Mitochondrial Nucleoids
Incomplete Mitophagy in Human Neutrophils Leads to Extrusion of Mitochondrial Nucleoids
Erin Quan Toyama,
Genomics Institute of the Novartis Research Foundation, USA
Identification of MFF as a Direct Substrate for AMPK
Identification of MFF as a Direct Substrate for AMPK
17:00—19:00
"Deathomics"
*
Matthew S. Bogyo,
Stanford University School of Medicine, USA
Jennie R. Lill,
Genentech, Inc., USA
Caspase Substrate Discovery
Caspase Substrate Discovery
James A. Wells,
University of California, San Francisco, USA
Caspase Kinetics
Caspase Kinetics
Harris G. Fienberg,
Stanford University, USA
Network Rewiring Is Critical for Non-Genetic Resistance to TRAIL
Network Rewiring Is Critical for Non-Genetic Resistance to TRAIL
James A. Clulow,
Imperial College London, UK
Short Talk: Unravelling the Targets of Electrophilic Natural Products using Quantitative Activity-Based Chemical Proteomics
Short Talk: Unravelling the Targets of Electrophilic Natural Products using Quantitative Activity-Based Chemical Proteomics
17:00—19:00
Mitochondria as Signaling Platform
*
Andrew G. Dillin,
University of California, Berkeley, USA
Marcia C. Haigis,
Harvard Medical School, USA
Mitochondrial Dynamics in Metabolic Adaptation
Mitochondrial Dynamics in Metabolic Adaptation
Zhijian "James" Chen,
University of Texas Southwestern Medical Center, USA
The Mitochondrial Pathway of Antiviral Innate Immune Response
The Mitochondrial Pathway of Antiviral Innate Immune Response
David C. Chan,
California Institute of Technology, USA
Molecular Regulation of Mitochondrial Dynamics
Molecular Regulation of Mitochondrial Dynamics
Andrea Rasola,
Università degli Studi di Padova, Italy
Short Talk: The Mitochondrial Chaperone TRAP1 and Neoplastic Transformation
Short Talk: The Mitochondrial Chaperone TRAP1 and Neoplastic Transformation
08:00—11:00
Post-Translational Control of Cell Death
*
Andreas E. Strasser,
Walter and Eliza Hall Institute of Medical Research, Australia
John Silke,
Walter and Eliza Hall Institute of Medical Research, Australia
cIAPs and Sharpin Regulate TNF/MLKL Dependent Necroptotic Cell Death and Developments in Targeting this Axis in Disease
cIAPs and Sharpin Regulate TNF/MLKL Dependent Necroptotic Cell Death and Developments in Targeting this Axis in Disease
Henning Walczak,
University College London, Cancer Institute, UK
New Traits of TRAIL in Cancer
New Traits of TRAIL in Cancer
Marion MacFarlane,
MRC Toxicology Unit, UK
Death Receptor Mechanisms: The ‘FLIP’ Side of the DISC
Death Receptor Mechanisms: The ‘FLIP’ Side of the DISC
Guy S. Salvesen,
Sanford-Burnham Medical Research Institute, USA
Proteolytic Crosstalk in Cell Death and Survival
Proteolytic Crosstalk in Cell Death and Survival
Yoshihisa Kaizuka,
National Institute for Materials Science, Japan
Short Talk: Signal Protein Clusters in Plasma Membranes Involved in Death Signaling and Adaptive Immunity
Short Talk: Signal Protein Clusters in Plasma Membranes Involved in Death Signaling and Adaptive Immunity
08:00—11:00
Quality Control
*
Richard J. Youle,
NINDS, National Institutes of Health, USA
Cole M. Haynes,
University of Massachusetts Medical School, USA
Coordinating Repair and Regeneration of Defective Mitochondrial via the UPRmt
Coordinating Repair and Regeneration of Defective Mitochondrial via the UPRmt
Jared Rutter,
University of Utah, USA
Functionalizing the Unannotated Mitochondrial Proteome
Functionalizing the Unannotated Mitochondrial Proteome
Dario C. Altieri,
Wistar Institute, USA
Mitochondrial Chaperones
Mitochondrial Chaperones
Giovanni Bénard,
INSERM, France
Short Talk: Mitochondrial Turnover and Energy Metabolism
Short Talk: Mitochondrial Turnover and Energy Metabolism
14:30—16:30
Workshop 2: RIP3/Necroptosis
*
Kim Newton,
Genentech, Inc., USA
Susana L. Orozco,
University of Washington, USA
RIPK1 both Positively and Negatively Regulates RIPK3 Oligomerization and Necroptosis.
RIPK1 both Positively and Negatively Regulates RIPK3 Oligomerization and Necroptosis.
Carlos F. Lopez,
Vanderbilt University, USA
Exploring how Cells Commit to Apoptotic or Necrotic Cell-Death
Exploring how Cells Commit to Apoptotic or Necrotic Cell-Death
Francis Ka-Ming Chan,
Duke University, USA
Necrotic and Non-Necrotic Functions of RIP3 in Injury-Induced Inflammation
Necrotic and Non-Necrotic Functions of RIP3 in Injury-Induced Inflammation
Mordechay Gerlic,
Sackler Faculty of Medicine, Tel Aviv University, Israel
RIPK1 Regulates Cell Death Driven Systemic Inflammation
RIPK1 Regulates Cell Death Driven Systemic Inflammation
17:00—19:00
Death Meets Survival
*
Douglas R. Green,
St. Jude Children's Research Hospital, USA
Pamela M. Holland,
Surface Oncology, USA
Death Receptor Agonists for Cancer: Which Is the Right TRAIL?
Death Receptor Agonists for Cancer: Which Is the Right TRAIL?
Marion C. Bonnet,
INSERM U976-Hopital St-Louis, France
Death and Survival of Keratinocytes
Death and Survival of Keratinocytes
Kim Newton,
Genentech, Inc., USA
Death by Kinases RIP1 and RIP3
Death by Kinases RIP1 and RIP3
Ben A. Croker,
Boston Children's Hospital, USA
Short Talk: Fas Controls Neutrophil Lifespan during Viral Infection and Is Negatively Regulated by TLR and IL-18 Signaling
Short Talk: Fas Controls Neutrophil Lifespan during Viral Infection and Is Negatively Regulated by TLR and IL-18 Signaling
17:00—19:00
Stem Cells and Development
*
Luca Scorrano,
University of Padova, Italy
Jahar Bhattacharya,
College of Physicians & Surgeons, Columbia University, USA
Mitochondrial Transfer from Bone-Marrow-Derived Stromal Cells to Pulmonary Alveoli Protects Against Acute Lung Injury
Mitochondrial Transfer from Bone-Marrow-Derived Stromal Cells to Pulmonary Alveoli Protects Against Acute Lung Injury
Carla Koehler,
University of California, Los Angeles, USA
Correcting Human Mitochondrial Mutations with Targeted RNA Import
Correcting Human Mitochondrial Mutations with Targeted RNA Import
Mireille Khacho,
University of Ottawa, Canada
Short Talk: Mitochondrial Dynamics in the Regulation of Stem Cell Maintenance and Cell Fate Decisions
Short Talk: Mitochondrial Dynamics in the Regulation of Stem Cell Maintenance and Cell Fate Decisions
Alison M. Burkart,
Joslin Diabetes Center, USA
Short Talk: Dissecting Relationships between Insulin Resistance and Mitochondrial Metabolism in Human iPS Cells
Short Talk: Dissecting Relationships between Insulin Resistance and Mitochondrial Metabolism in Human iPS Cells
Konstanze F. Winklhofer,
Ruhr University Bochum, Germany
Short Talk: Talk Title to be Announced
Short Talk: Talk Title to be Announced
Michael A. Frohman,
Stony Brook University, USA
Roles for the Lipid-Signaling Enzymes MitoPLD and Lipin 1 in Mitochondrial Dynamics, piRNA Biogenesis, and Spermatogenesis
Roles for the Lipid-Signaling Enzymes MitoPLD and Lipin 1 in Mitochondrial Dynamics, piRNA Biogenesis, and Spermatogenesis
08:00—11:15
Leveraging Model Organisms
*
Marion MacFarlane,
MRC Toxicology Unit, UK
Eli Arama,
Weizmann Institute of Science, Israel
A Mitochondrial-Based Rate-Limiting Mechanism for Caspase Activation during Sperm Differentiation in Drosophila
A Mitochondrial-Based Rate-Limiting Mechanism for Caspase Activation during Sperm Differentiation in Drosophila
Hyung Don Ryoo,
New York University Langone Medical Center, USA
Regulating the Subcellular Distribution of a Pro-Apoptotic Protein, Hid
Regulating the Subcellular Distribution of a Pro-Apoptotic Protein, Hid
Eric H. Baehrecke,
University of Massachusetts Medical School, USA
Regulation and Function of Autophagy during Cell Death
Regulation and Function of Autophagy during Cell Death
Keren Yacobi Sharon,
Weizmann Institute of Science, Israel
Short Talk: Germ Cell Death: A Physiological Alternative Cell Death Pathway in Drosophila
Short Talk: Germ Cell Death: A Physiological Alternative Cell Death Pathway in Drosophila
Barbara Conradt,
Ludwig-Maximilians-Universitat, Germany
Short Talk: C. elegans CED-3 Caspase Regulates Centrosome Asymmetry in an Apoptotic Death
Short Talk: C. elegans CED-3 Caspase Regulates Centrosome Asymmetry in an Apoptotic Death
08:00—11:00
Environmental Control of Mitochondrial Physiology
Andrew G. Dillin,
University of California, Berkeley, USA
The Conserved Histone Lysine Demethylase PHF8 Regulates Mitochondrial ETC-Mediated Longevity
The Conserved Histone Lysine Demethylase PHF8 Regulates Mitochondrial ETC-Mediated Longevity
*
Lluis Fajas,
Université de Lausanne, Switzerland
Participation of CDK4 in the Regulation of Mitochondrial Metabolism and Energy Homeostasis
Participation of CDK4 in the Regulation of Mitochondrial Metabolism and Energy Homeostasis
Erika L. Pearce,
Max Planck Institute of Immunobiology and Epigenetics, Germany
Lipid Metabolism, Mitochondria, and Memory T Cell Generation
Lipid Metabolism, Mitochondria, and Memory T Cell Generation
Sameer Kulkarni,
Nestlè Institute of Health Sciences SA, Switzerland
Short Talk: Impact of Liver-Specific Deletion of Mfn1 and Mfn2 in Metabolic Health
Short Talk: Impact of Liver-Specific Deletion of Mfn1 and Mfn2 in Metabolic Health
14:30—16:30
Workshop 2: Emerging Topics in Mitochondrial Dynamics and Physiology
*
Cole M. Haynes,
University of Massachusetts Medical School, USA
Mariusz Karbowski,
Amgen Inc, USA
MARCH5-Dependent Regulation of the OMM-Associated Degradation (OMMAD) Pathway and Mitochondrial Steps in Apoptosis
MARCH5-Dependent Regulation of the OMM-Associated Degradation (OMMAD) Pathway and Mitochondrial Steps in Apoptosis
Adam L. Hughes,
University of Utah, USA
An Autophagy-Dependent Pathway for Removal of Individual Proteins from Dysfunctional Mitochondria
An Autophagy-Dependent Pathway for Removal of Individual Proteins from Dysfunctional Mitochondria
Noriyuki Matsuda,
Tokyo Metropolitan Institute of Medical Science, Japan
Identification of the Genuine Substrate of PINK1 that Activates Parkin
Identification of the Genuine Substrate of PINK1 that Activates Parkin
Christof Osman,
University of California, San Francisco, USA
Live-Cell Microscopy of Mitochondrial DNA Suggests a Mechanism for its Inheritance and Distribution
Live-Cell Microscopy of Mitochondrial DNA Suggests a Mechanism for its Inheritance and Distribution
A. Phillip West,
Yale University School of Medicine, USA
Altered Mitochondrial DNA Dynamics Elicits a Cell-Intrinsic Antiviral Signaling Program
Altered Mitochondrial DNA Dynamics Elicits a Cell-Intrinsic Antiviral Signaling Program
Atsushi Tanaka,
Yamagata University, Japan
Mechanisms and Process of Mitochondrial Collapse in Autophagy-Deficient Mice
Mechanisms and Process of Mitochondrial Collapse in Autophagy-Deficient Mice
Brian Alexander Roelofs,
University of Maryland Baltimore, USA
Npl4 Is Required for p97 to Perform Mitochondrial Quality Control Functions
Npl4 Is Required for p97 to Perform Mitochondrial Quality Control Functions
17:00—19:00
Systems Biology and Death Imaging
*
Guy S. Salvesen,
Sanford-Burnham Medical Research Institute, USA
Jessie Ochoa,
University of California, Santa Cruz, USA
Short Talk: Cytological Profiling of Natural Products to Identify Modes of Action
Short Talk: Cytological Profiling of Natural Products to Identify Modes of Action
Sally A. Kornbluth,
Duke University Medical Center, USA
Control of Caspase 2 Activation
Control of Caspase 2 Activation
Pascal Meier,
Institute of Cancer Research, UK
Regulation of the Ubiquitin E3 Ligase cIAP1
Regulation of the Ubiquitin E3 Ligase cIAP1
17:00—19:00
Mitochondria in Tissue Homeostasis
*
Jared Rutter,
University of Utah, USA
Eric A. Shoubridge,
McGill University, Canada
Posttranscriptional Regulation of Mitochondrial Gene Expression
Posttranscriptional Regulation of Mitochondrial Gene Expression
Rodrigue Rossignol,
University of Bordeaux, France
Oncogenic RAS Inhibits the LKB1-AMPK Axis and Repatterns Energy Metabolism
Oncogenic RAS Inhibits the LKB1-AMPK Axis and Repatterns Energy Metabolism
Ralph J. DeBerardinis,
University of Texas Southwestern Medical Center, USA
Mitochondrial Metabolism in Cancer
Mitochondrial Metabolism in Cancer
Dongryeol Ryu,
Pusan National University, South Korea
Short Talk: SIRT7 Regulates Mitochondrial Homeostasis via the Deacetylation and Activation of GABPbeta1
Short Talk: SIRT7 Regulates Mitochondrial Homeostasis via the Deacetylation and Activation of GABPbeta1
*Session Chair †Invited, not yet responded.
We gratefully acknowledge support for this conference from:
Keystone Symposia thanks our Sponsors for generously supporting this meeting:
|
|
We gratefully acknowledge additional in-kind support for this conference from those foregoing speaker expense reimbursements:
We appreciate the organizations that provide Keystone Symposia with additional support, such as marketing and advertising:
|
|
Special thanks to the following for their support of Keystone Symposia initiatives to increase participation at this meeting by scientists from underrepresented backgrounds:
Click here to view more of these organizations
|
If your organization is interested in joining these entities in support of Keystone
Symposia, please contact: Sarah Lavicka,
Director of Corporate Relations, Email: sarahl@keystonesymposia.org, Phone:+1 970-262-2690 Click here for more information on Industry Support and Recognition Opportunities. If you are interested in becoming an advertising/marketing in-kind partner, please contact: Nick Dua, Senior Director, Communications, Email: nickd@keystonesymposia.org, Phone:+1 970-262-1179 |
